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A Mind is a Terrible Thing to Waste

Peculiar how I get called a “troll” in “political discussion” groups when the reality is I’m not embarrassed to interrupt a collectivist/totalitarian confirmation bias circle-jerk and explain to the domesticated life-long child sheeple flock why a mind is a terrible thing to waste and somewhere below all the indoctrination the human they were born to be is trying to get out.

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How the Tyranny of the Ruling Classes is Greatly Supported by Tyranny by the Ruled (Michael-Tsarion-Psycopathy-Identification.avi)


How the Tyranny of the Ruling Classes is Greatly Supported by Tyranny by the Ruled

One of many examples is politician/royalty worship and those sickos as role models.

How do you treat the nonhuman animals living with you or who have lived with you, and the same for other beings such as children in the same predicament/situation?

“The whole point is we cling like monkeys to the rotten sweets when the gooey good stuff is being offered to us, and this pathological identification with their [Big Brother’s] thinking…it’s happened subconsciously…how you disempower yourself on a daily basis…become still…so you can observe, carefully, what you’re doing to yourself and to other people…that’s when Big Brother’s eyes open…they have a whole Leviathan to get you out of that center…there is no A to Z, this is the whole essence of selfhood, only you can walk your path for you. The only message we can give you is to walk it fearlessly.”

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Ninety References Related to Crime Families

bush-clinton-topperMy point isn’t that some people have a moral high ground over others; it’s that the brain is an organ and just like the heart, pancreas, or any other organ and based on both nature and nurture is often predisposed — not predestined, predisposed, aka “potential” — to unhealthy behaviors. Rather than alleged moral high grounds such as political correctness filtering which scientific facts get acknowledged, individuals can have significantly better qualities of life the more they know about their nature and nurture background and those of people in each others’ lives.

The book Evil Genes by Barbara Oakley has 12 chapters, and Chapter 3 with 54 references and Chapter 4 with 36 references focus more on biology than the other chapters:


1 Robert Plomin, Nature and Nurture: An Introduction to Human Behavioral Genetics (Belmont, CA: Wadworth/Thomson Learning, 2004), p. 67. See also Lisabeth F. DiLalla and Irving I. Gottesman, Behavior Genetics Principles, 1st ed. (Washington, DC: American Psychological Association, 2004).

2 Plomin, Nature and Nurture, p. 112. L. B. Koenig et al., “Genetic and Environmental Influences on Religiousness: Findings for Retrospective and Current Religiousness Ratings,” Journal of Personality 73, no. 2 (2005): 471-88.

3 K. P. Harden et al., “Marital Conflict and Conduct Disorder in Children-of-Twins,” Child Development 78, no. 1 (2007): in press.

4 Joshua Roffman et al., “Neuroimaging-Genetic Paradigms: A New Approach to Investigate the Pathophysiology and Treatment of Cognitive Deficits in Schizophrenia,” Harvard Review of Psychiatry 14, no. 2 (2006): 78-91.

5 Richard Redon et al., “Global Variation in Copy Number in the Human Genome,” Nature 444, no. 7118 (2006): 444-54.

6 David J. Buller, Adapting Minds: Evolutionary Psychology and the Persistent Quest for Human Nature (Cambridge, MA: MIT Press, 2005), p. 437.

7 E. Meshorer et al., “SC35 Promotes Sustainable Stress-Induced Alternative Splicing of Neuronal Acetylcholinesterase mRNA,” Molecular Psychiatry 10, no. 11 (2005): 985-97; E. Meshorer and H. Soreq, “Virtues and Woes of AChE Alternative Splicing in Stress-Related Neuropathologies,” Trends in Neurosciences 29, no. 4 (2006): 216-24; R. Valgardsdottir et al., “Structural and Functional Characterization of Noncoding Repetitive RNAs Transcribed in Stressed Human Cells,” Molecular Biology of the Cell 16, no. 6 (2005): 2597-2604.

8 John Rose, Human Stress and the Environment: Health Aspects (Taylor & Francis, 1994), pp. 1, 8, 133.

9 Sridhar Prathikanti and Daniel R. Weinberger, “Psychiatric Genetic —the New Era: Genetic Research and Some Clinical Implications,” British Medical Bulletin 73 and 74 (2005): 107-22. But see Robert Plomin, “Finding Genes in Child Psychology and Psychiatry: When Are We Going to Be There?” Journal of Child Psychology and Psychiatry 46, no. 10 (2005): 1030-38, for skepticism regarding this approach.

10 Daniel Weinberger, in discussion with the author, June 1, 2006.

11 C. H. Chen et al., “Brain Imaging Correlates of Depressive Symptom Severity and Predictors of Symptom Improvement after Antidepressant Treatment,” Biological Psychiatry [Epub ahead of print] (2007).

12 J. L. Roffman et al., “Neuroimaging and the Functional Neuroanatomy of Psychotherapy,” Psychological Medicine 35, no. 10 (2005): 1385-98.

13 Weinberger, in discussion with the author, June 1, 2006.

14 D. L. Murphy et al., “Brain Serotonin Neurotransmission,” Journal of Clinical Psychiatry 59, Suppl

15 (1998): 4-12. 15 Robert Plomin, M. J. Owen, and P. McGuffin, “The Genetic Basis of Complex Human Behaviors,” Science 264, no. 1733-39 (1994); A. Reif and K. P. Lesch, “Toward a Molecular Architecture of Personality,” Behavioural Brain Research 139, no. 1-2 (2003): 1-20.

16 Berend Olivier, “Serotonin and Aggression,” Annals of the New York Academy of Sciences 1036 (2004): 382-92.

17 A. S. New et al., “Impulsive Aggression Associated with HTR1B Genotype in Personality Disorder,” in Annual Meeting of the American Psychiatric Association NR 388 (1999); A. S. New et al., “Serotonin Related Genotype and Impulsive Aggression,” in Annual Meeting of the Society of Biological Psychiatry 45, Abstract #387 (1999); F. Rybakowski et al., “The 5-HT2A-1438 A/G and 5-HTTLPR Polymorphisms and Personality Dimensions in Adolescent Anorexia Nervosa: Association Study,” Neuropsychobiology 53, no. 1 (2006): 33-39.

18 T. Iidaka et al., “A Variant C178T in the Regulatory Region of the Serotonin Receptor Gene HTR3A Modulates Neural Activation in the Human Amygdala,” Journal of Neuroscience 25, no. 27 (2005): 6460-66.

19 X. Ni et al., “Association between Serotonin Transporter Gene and Borderline Personality Disorder,” Journal of Psychiatric Research 40, no. 5 (2006): 448-53; L. Pezawas et al., “5-HTTLPR Polymorphism Impacts Human Cingulate-Amygdala Interactions: A Genetic Susceptibility Mechanism for Depression,” Nature Neuroscience 8, no. 6 (2005): 828-34; H. Steiger et al., “The 5HTTLPR Polymorphism, Psychopathologic Symptoms, and Platelet [3H-] Paroxetine Binding in Bulimic Syndromes,” International Journal of Eating Disorders 37, no. 1 (2005): 57-60.

20 Ahmad R. Hariri et al., “A Susceptibility Gene for Affective Disorders and the Response of the Human Amygdala,” Archives of General Psychiatry 62, no. 2 (2005): 146-54; Pezawas et al., “5-HTTLPR.”

21 S. Eddahibi et al., “Serotonin Transporter Overexpression Is Responsible for Pulmonary Artery Smooth Muscle Hyperplasia in Primary Pulmonary Hypertension,” Journal of Clinical Investigation 108, no. 8 (2001): 1141-50; S. Eddahibi et al., “Hyperplasia of Pulmonary Artery Smooth Muscle Cells Is Causally Related to Overexpression of the Serotonin Transporter in Primary Pulmonary Hypertension,” Chest 121, no. 3 (2002): 97S-98S.

22 R. Cacabelos et al., “Molecular Genetics of Alzheimer’s Disease and Aging,” Methods and Findings in Experimental Clinical Pharmacology 27, no. Suppl A (2005): 1-573; D. K. Lahiri, C. Ghosh, and Y. W. Ge, “A Proximal Gene Promoter Region for the Beta-Amyloid Precursor Protein Provides a Link between Development, Apoptosis, and Alzheimer’s Disease,” Annals of the New York Academy of Sciences 1010 (2003): 643-47.

23 Reinaldo B. Oriá et al., “Role of Apolipoprotein E4 in Protecting Children against Early Childhood Diarrhea Outcomes and Implications for Later Development,” Medical Hypotheses 68, no. 5 (2007): 1099-1107.

24 Christian R. A. Mondadori et al., “Better Memory and Neural Efficiency in Young Apolipoprotein Eε4 Carriers,” Cerebral Cortex, epub ahead of print (2006): bhl103.

25 Nancy Touchette, “Gene Variation Affects Memory,” Genome News Network, 2003, http://www.genomenewsnetwork.org/articles/08_03/memory.shtml (accessed May 11, 2006).

26 S. Sen et al., “A BDNF Coding Variant Is Associated with the NEO Personality Inventory Domain Neuroticism, a Risk Factor for Depression,” Neuropsychopharmacology 28, no. 2 (2003): 397-401.

27 Jim Phelps, “Connecting Anxiety and Depression via the Serotonin Transporter Gene,” PsychEducation.org: Extensive Mental Health Information on Specific Topics, 2006, http://www.psycheducation.org/mechanism/4WhyShortsLongs.htm (accessed July 10, 2006).

28 M. Ribases et al., “Contribution of NTRK2 to the Genetic Susceptibility to Anorexia Nervosa, Harm Avoidance and Minimum Body Mass Index,” Molecular Psychiatry 10, no. 9 (2005): 851-60.

29 J. Strauss et al., “BDNF and COMT Polymorphisms: Relation to Memory Phenotypes in Young Adults with Childhood-Onset Mood Disorder,” Neuromolecular Medicine 5, no. 3 (2004): 181-92.

30 A. Thapar et al., “Catechol O-methyltransferase Gene Variant and Birth Weight Predict Early-Onset Antisocial Behavior in Children with Attention-Deficit/Hyperactivity Disorder,” Archives of General Psychiatry 62, no. 11 (2005): 1275-78.

31 Ke Xu and D. Goldman, “Catechol-O-methyltransferase Genotype, Intermediate Phenotype, and Psychiatric Disorders,” in Cell Biology of Addiction, ed. Bertha Madras et al. (Cold Spring Harbor, NY: Cold Spring Harbor Laboratory Press, 2005), pp. 29-44.

32 K. Xu, M. Ernst, and D. Goldman, “Imaging Genomics Applied to Anxiety, Stress Response, and Resiliency,” Neuroinformatics 4, no. 1 (2006): 51-64.

33 Xu and Goldman, “Catechol-O-methyltransferase Genotype.”

34 Michael N. Smolka et al., “Catechol-O-methyltransferase val158met Genotype Affects Processing of Emotional Stimuli in the Amygdala and Prefrontal Cortex,” Journal of Neuroscience 25, no. 4 (2005): 836-43.

35 Ibid.; Xu, Ernst, and Goldman, “Imaging Genomics.”

36 Xu, Ernst, and Goldman, “Imaging Genomics.”

37 I. W. Craig, “The Role of Monoamine Oxidase A, MAOA, in the Aetiology of Antisocial Behaviour: The Importance of Gene-Environment Interactions,” Novartis Foundation Symposium 268 (2005): 227-37; discussion 237-41, 242-53; A. Serretti et al., “Temperament and Character in Mood Disorders: Influence of DRD4, SERTPR, TPH and MAO-A Polymorphisms,” Neuropsychobiology 53, no. 1 (2006): 9-16.

38 Christian P. Jacob et al., “Cluster B Personality Disorders Are Associated with Allelic Variation of Monoamine Oxidase A Activity,” Neuropsychopharmacology 30, no. 9 (2005): 1711-18.

39 Andreas Meyer-Lindenberg et al., “Neural Mechanisms of Genetic Risk for Impulsivity and Violence in Humans,” Proceedings of the National Academy of Sciences 103, no. 16 (2006): 6269-74.

40 Ibid.

41 H. G. Brunner et al., “Abnormal Behavior Associated with a Point Mutation in the Structural Gene for Monoamine Oxidase A,” Science 262, no. 5133 (1993): 578-80.

42 Avshalom Caspi et al., “Role of Genotype in the Cycle of Violence in Maltreated Children,” Science 297 (2002): 851-54.

43 Meshorer et al., “SC35”; Meshorer and Soreq, “Virtues and Woes.”

44 T. E. Moffitt, “The New Look of Behavioral Genetics in Developmental Psychopathology: Gene-Environment Interplay in Antisocial Behaviors,” Psychological Bulletin 131, no. 4 (2005): 533-54.

45 S. M. Brown et al., “A Regulatory Variant of the Human Tryptophan Hydroxylase-2 Gene Biases Amygdala Reactivity,” Molecular Psychiatry 10, no. 9 (2005): 884-88, 805; Serretti et al., “Temperament”; G. Zaboli et al., “Tryptophan Hydroxylase-1 Gene Variants Associate with a Group of Suicidal Borderline Women,” Neuropsychopharmacology 31, no. 9 (2006): 1982-90.

46 J. Auerbach et al., “Dopamine D4 Receptor (D4DR) and Serotonin Transporter (5-HTTLPR) Polymorphisms in the Determination of Temperament in 2-Month-Old Infants,” Molecular Biology 4 (1999): 369-73.

47 “Common Gene Version Optimizes Thinking—but with a Possible Downside,” NIH News, February 8, 2007, http://www.nih.gov/news/pr/feb2007/nimh-08.htm (accessed February 25, 2007).

48 D. T. Lykken et al., “Emergenesis: Genetic Traits That May Not Run in Families,” American Psychologist 47, no. 12 (1992): 1565-77.

49 R. Bachner-Melman et al., “AVPR1a and SLC6A4 Gene Polymorphisms Are Associated with Creative Dance Performance,” PLoS Genetics 1, no. 3 (2005): e42; R. Oerter, “Biological and Psychological Correlates of Exceptional Performance in Development,” Annals of the New York Academy of Sciences 999 (2003): 451-60.

50 E. F. Torrey and R. H. Yolken, “Toxoplasma gondii and Schizophrenia,” Emerging Infectious Diseases 9, no. 11 (2003): 1375-80.

51 Ni et al., “Association between Serotonin” ; A. E. Skodol et al., “The Borderline Diagnosis II: Biology, Genetics, and Clinical Course,” Biological Psychiatry 51, no. 12 (2002): 951-63.

52 Larry J. Siever, Harold W. Koenigsberg, and Deidre Reynolds, “Neurobiology of Personality Disorders: Implications for a Neurodevelopmental Model,” in Neurodevelopmental Mechanisms in Psychopathology, ed. Dante Cicchetti and Elaine Walker (New York: Cambridge University Press, 2003), pp. 416-17; A. E. Skodol and Donna S. Bender, “Why Are Women Diagnosed Borderline More Than Men?” Psychiatric Quarterly 74, no. 4 (2003): 349-60; S. Torgersen, “Genetics in Borderline Conditions,” Acta Psychiatrica Scandinavica Supplementum 379 (1994): 19-25.

53 Norbert Wiener, Ex-Prodigy: My Childhood and Youth (Cambridge, MA: MIT Press, 1953), p. 158.

54 Ibid., pp. 11-12. 55 Nicholas Wade, “Researchers Say Intelligence and Diseases May Be Linked in Ashkenazic Genes,” New York Times, June 3, 2005.


1 “Functional Families, Dysfunctional Brains,” Science Daily, April 10, 1998, http://www.sciencedaily.com/releases/1998/04/980410101830.htm (accessed November 24, 2005).

2 An excellent article about both the strengths and weaknesses of imaging approaches can be found in Malcolm Gladwell, “The Picture Problem: Mammography, Air Power, and the Limits of Looking,” New Yorker, December 13, 2004.

3 Kent A. Kiehl et al., “Limbic Abnormalities in Affective Processing by Criminal Psychopaths as Revealed by Functional Magnetic Resonance Imaging,” Biological Psychiatry 50, no. 9 (2001): 677-84.

4 S. W. Anderson et al., “Impairment of Social and Moral Behavior Related to Early Damage in Human Prefrontal Cortex,” Nature Neuroscience 2 (1999): 1032-37; A. Bechara et al., “Insensitivity to Future Consequences Following Damage to Human Prefrontal Cortex,” Cognition 50 (1994): 7-15; A. R. Damasio, D. Tranel, and H. Damasio, “Individuals with Sociopathic Behavior Caused by Frontal Damage Fail to Respond Autonomically to Social Stimuli,” Behavioural Brain Research 41 (1990): 81-94.

5 Kent Kiehl in communication with the author, July 5, 2005.

6 Adrian Raine et al., “Corpus Callosum Abnormalities in Psychopathic Antisocial Individuals,” Archives of General Psychiatry 60 (2003): 1134-42.

7 Adrian Raine and Yaling Yang, “The Neuroanatomical Bases of Psychopathy : A Review of Brain Imaging Findings,” in Handbook of Psychopathy, ed. Christopher J. Patrick (New York: Guilford Press, 2006), pp. 278-95.

8 G. P. Shumyatsky et al., “Stathmin, a Gene Enriched in the Amygdala, Controls Both Learned and Innate Fear,” Cell 123, no. 4 (2005): 697-709.

9 James Blair, Derek Mitchell, and Karina Blair, The Psychopath: Emotion and the Brain (Malden, MA: Blackwell Publishing, 2005), p. 139; R. J. Blair, “Neurobiological Basis of Psychopathy,” British Journal of Psychiatry 182 (2003): 5-7; F. Schneider et al., “Functional Imaging of Conditioned Aversive Emotional Responses in Antisocial Personality Disorder,” Neuropsychobiology 42 (2000): 192-201.

10 Sharon Ishikawa and Adrian Raine, “Prefrontal Deficits and Antisocial Behavior: A Causal Model,” in Causes of Conduct Disorder and Juvenile Delinquency, ed. Avshalom Caspi, Benjamin B. Lahey, and Terrie Moffitt (New York: Guilford Press, 2003), pp. 277-304.

11 “Functional Families, Dysfunctional Brains.”

12 Adrian Raine et al., “Reduced Prefrontal and Increased Subcortical Brain Functioning Assessed Using Positron Emission Tomography in Predatory and Affective Murderers,” Behavioral Sciences and the Law 16 (1998): 319-32.

13 Bechara et al., “Insensitivity” ; A. Bechara et al., “Different Contributions of the Human Amygdala and Ventromedial Prefrontal Cortex to Decision-Making,” Journal of Neuroscience 19, no. 13 (1999): 5473-81; L. K. Fellows and M. J. Farah, “Different Underlying Impairments in Decision-Making Following Ventromedial and Dorsolateral Frontal Lobe Damage in Humans,” Cerebral Cortex 15, no. 1 (2005): 58-63.

14 This example is after that of Meloy, Violence.

15 Adrian Raine, “Psychopathy, Violence, and Brain Imaging,” in Violence and Psychopathy, ed. Adrian Raine and José Sanmartín (New York: Kluwer Academic/Plenum Publishers, 2001), pp. 35-56.

16 Adrian Raine, “Murderous Minds: Can We See the Mark of Cain?” Cerebrum 1, no. 1 (1999): 15-30.

17 Kent A. Kiehl et al., “Temporal Lobe Abnormalities in Semantic Processing by Criminal Psychopaths as Revealed by Functional Magnetic Resonance Imaging,” Psychiatry Research: Neuroimaging 130, no. 1 (2004): 27-42.

18 Terrence W. Deacon, The Symbolic Species (New York: W. W. Norton & Company, 1997), pp. 427, 459; Andreas Meyer-Lindenberg et al., “Neural Correlates of Genetically Abnormal Social Cognition in Williams Syndrome,” Nature Neuroscience 8, no. 8 (2005): 991-95.

19 David Dobbs, “The Gregarious Brain,” New York Times Magazine, July 8, 2007.

20 Jef Allbright, “Scientists Watch the Brain Wrestle with Moral Dilemmas,” Jef’s Web Files, 2004, http://www.jefallbright.net/node/2691 (accessed December 29, 2005); Jorge Moll et al., “The Neural Correlates of Moral Sensitivity: A Functional Magnetic Resonance Imaging Investigation of Basic and Moral Emotions,” Journal of Neuroscience 22, no. 7 (2002): 2730-36.

21 Dharol Tankersley, C. Jill Stowe, and Scott A. Huettel, “Altruism Is Associated with an Increased Neural Response to Agency,” Nature Neuroscience 10 (2007): 150-51.

22 E. J. Mundell, “Why Do Good? Brain Study Offers Clues,” HealthDay, January 22, 2007, http://www.healthday.com/Article.asp?AID=601147 (accessed January 23, 2007).

23 H. Takahashi et al., “Brain Activation Associated with Evaluative Processes of Guilt and Embarrassment: An fMRI Study,” Neuroimage 23, no. 3 (2004): 967-74. See also S. Berthoz et al., “Affective Response to One’s Own Moral Violations,” Neuroimage 31, no. 2 (2006): 945-50; E. C. Finger et al., “Caught in the Act: The Impact of Audience on the Neural Response to Morally and Socially Inappropriate Behavior,” Neuroimage 33, no. 1 (2006): 414-21; C. L. Harenski and S. Hamann, “Neural Correlates of Regulating Negative Emotions Related to Moral Violations,” Neuroimage 30, no. 1 (2006): 313-24; Moll, Oliveira-Sousa, and Eslinger, “Morals and the Human Brain.”

24 D. J. Stein and D. Kaminer, “Forgiveness and Psychopathology: Psychobiological and Evolutionary Underpinnings,” CNS Spectrums 11, no. 2 (2006): 87-89.

25 Raine and Yang, “Neural Foundations.” This paper contains a comprehensive review of virtually all neurological imaging findings related to antisocial behavior, which is much more extensive than the basic findings presented here.

26 Paul J. Frick and Monica A. Marsee, “Psychopathy and Developmental Pathways to Antisocial Behavior in Youth,” in Handbook of Psychopathy, ed. Christopher J. Patrick (New York: Guilford Press, 2005), pp. 353-74.

27 Meloy, Violence, pp. 121-22.

28 “Psychologist Adds Scientific Insight to Loaded Label of ‘Psychopath, ’” Physorg.com, June 28, 2006, http://www.physorg.com/news70728146.html (accessed July 1, 2006).

29 Joseph Newman, in correspondence with the author, February 1, 2007.

30 Sandra Blakeslee, “Cells That Read Minds,” New York Times, January 10, 2006.

31 R. James Blair and Karina S. Perschardt, “Empathy: A Unitary Circuit or a Set of Dissociable Neuro-Cognitive Systems?” Behavioral and Brain Sciences 25 (2002): 27-28.

32 Deacon, Symbolic Species, p. 402; Linda Mealey and Stuart Kinner, “The Perception-Action Model of Empathy and Psychopathic ‘Cold-Heartedness, ’” Behavioral and Brain Sciences 25 (2002): 42-43.

33 Raine and Yang, “Neuroanatomical Bases”; Yaling Yang et al., “Volume Reduction in Prefrontal Gray Matter in Unsuccessful Criminal Psychopaths,” Biological Psychiatry 57, no. 10 (2005): 1103-1108.

34 Jamie Talan, “Lying Liars,” Scientific American Mind (April 2006): 8; Y. Yang et al., “Prefrontal White Matter in Pathological Liars,” British Journal of Psychiatry 187 (2005): 320-25.

35 Lisa Desai, “‘Corporate Psychopaths’ at Large,” CNN, August 26, 2004, http://edition.cnn.com/2004/BUSINESS/08/26/corporate.psychopaths/ (accessed September 28, 2005). See also Paul Babiak and Robert Hare, Snakes in Suits: When Psychopaths Go to Work (New York: HarperCollins, 2006).

36 Desai, “‘Corporate Psychopaths’ at Large.”

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My Relationship Line in the Sand aka I No Longer Do Crazy

My Relationship Line in the Sand aka I No Longer Do Crazy

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